Zinc deficiency causes neural tube defects through attenuation of p53 ubiquitylation
Journal Article
Overview
publication date
- December 15, 2018
has subject area
- Animals
- Apoptosis
- Biochemical Phenomena - Signal Transduction
- Biochemical Phenomena - Transcription, Genetic
- Biochemical Phenomena - Ubiquitination
- Biochemical Phenomena - Ubiquitination
- Carrier Proteins - Tumor Suppressor Protein p53
- Cell Line
- Cell Physiological Phenomena - Signal Transduction
- Cells - Neuroepithelial Cells
- Cells - Neuroepithelial Cells
- Congenital Abnormalities - Neural Tube Defects
- DNA-Binding Proteins - Tumor Suppressor Protein p53
- Elements - Zinc
- Epithelial Cells - Neuroepithelial Cells
- Gene Expression - Transcription, Genetic
- Gene Expression Regulation - Ubiquitination
- Metabolism - Ubiquitination
- Metals - Zinc
- Mice
- Models, Biological
- Nervous System - Neuroepithelial Cells
- Nervous System - Neuroepithelial Cells
- Nervous System Diseases - Neural Tube Defects
- Neural Tube
- Nuclear Proteins - Tumor Suppressor Protein p53
- Organelles - Mitochondria
- Peripheral Nervous System - Neuroepithelial Cells
- Peripheral Nervous System - Neuroepithelial Cells
- Phosphoproteins - Tumor Suppressor Protein p53
- Subcellular Fractions - Mitochondria
- Transcriptional Activation
- Transition Elements - Zinc
- Tumor Suppressor Proteins - Tumor Suppressor Protein p53
has restriction
- green
Date in CU Experts
- December 19, 2018 6:46 AM
Full Author List
- Li H; Zhang J; Niswander L
author count
- 3
citation count
- 25
published in
- Development (Cambridge) Journal
Other Profiles
International Standard Serial Number (ISSN)
- 0950-1991
Electronic International Standard Serial Number (EISSN)
- 1477-9129
Digital Object Identifier (DOI)
Additional Document Info
volume
- 145
issue
- 24
number
- ARTN dev169797