The maternal interleukin-17a pathway in mice promotes autism-like phenotypes in offspring
Journal Article
Overview
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Animals
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Autism Spectrum Disorder
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Behavior, Animal
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Behavioral Symptoms
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Biochemical Phenomena - Signal Transduction
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Biological Factors - Interleukin-17
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Blood Proteins - Antibodies, Blocking
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Cell Physiological Phenomena - Signal Transduction
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Cells - Th17 Cells
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Cells - Th17 Cells
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Cells - Th17 Cells
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Cerebral Cortex
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DNA-Binding Proteins - Nuclear Receptor Subfamily 1, Group F, Member 3
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Female
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Globulins - Antibodies, Blocking
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Hemic and Immune Systems - Th17 Cells
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Hemic and Immune Systems - Th17 Cells
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Hemic and Immune Systems - Th17 Cells
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Immune System - Th17 Cells
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Immune System - Th17 Cells
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Immune System - Th17 Cells
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Immunoproteins - Antibodies, Blocking
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Male
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Maternal-Fetal Exchange
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Mice
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Mutation
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Peptides - Interleukin-17
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Phenotype
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Pregnancy
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Prenatal Exposure Delayed Effects
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Proteins - Interleukin-17
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RNA, Messenger
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Receptors, Cytoplasmic and Nuclear - Nuclear Receptor Subfamily 1, Group F, Member 3
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Receptors, Cytoplasmic and Nuclear - Receptors, Retinoic Acid
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Transcription Factors - Receptors, Retinoic Acid
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Date in CU Experts
Full Author List
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Choi GB; Yim YS; Wong H; Kim S; Kim H; Kim SV; Hoeffer CA; Littman DR; Huh JR
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