Compound Deficiencies in Multiple Fibroblast Growth Factor Signalling Components Differentially Impact the Murine Gonadotrophin-Releasing Hormone System
Journal Article
Overview
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Animal Population Groups - Mice, Transgenic
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Animals
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Animals, Inbred Strains - Mice, Inbred C57BL
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Biochemical Phenomena - Signal Transduction
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Biological Factors - Fibroblast Growth Factor 8
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Cell Physiological Phenomena - Signal Transduction
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Cells - Neurons
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Congenital Abnormalities - Kallmann Syndrome
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Diencephalon - Hypothalamus
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Female Urogenital Diseases - Kallmann Syndrome
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Genetic Diseases, Inborn - Kallmann Syndrome
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Gonadal Disorders - Kallmann Syndrome
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Hormones, Hormone Substitutes, and Hormone Antagonists - Gonadotropin-Releasing Hormone
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Humans
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Hypogonadism
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Hypogonadism - Kallmann Syndrome
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Limbic System - Hypothalamus
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Male Urogenital Diseases - Kallmann Syndrome
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Mice
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Mice - Mice, Inbred C57BL
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Mice - Mice, Transgenic
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Nerve Tissue Proteins - Gonadotropin-Releasing Hormone
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Nervous System - Neurons
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Oligopeptides - Gonadotropin-Releasing Hormone
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Peptides - Fibroblast Growth Factor 8
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Peptides - Gonadotropin-Releasing Hormone
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Peptides - Gonadotropin-Releasing Hormone
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Protein-Tyrosine Kinases - Receptor, Fibroblast Growth Factor, Type 1
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Protein-Tyrosine Kinases - Receptor, Fibroblast Growth Factor, Type 3
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Proteins - Fibroblast Growth Factor 8
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Proto-Oncogene Proteins - Receptor, Fibroblast Growth Factor, Type 3
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Receptors, Cell Surface - Receptor, Fibroblast Growth Factor, Type 1
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Receptors, Cell Surface - Receptor, Fibroblast Growth Factor, Type 3
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Receptors, Fibroblast Growth Factor - Receptor, Fibroblast Growth Factor, Type 1
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Receptors, Fibroblast Growth Factor - Receptor, Fibroblast Growth Factor, Type 3
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Date in CU Experts
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September 9, 2013 10:25 AM
Full Author List
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Chung WCJ; Matthews TA; Tata BK; Tsai P-S
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