Disordered hinge regions of the AP-3 adaptor complex promote vesicle budding from the late Golgi in yeast. Journal Article uri icon

Overview

abstract

  • Vesicles bud from maturing Golgi cisternae in a programmed sequence. Budding is mediated by adaptors that recruit cargoes and facilitate vesicle biogenesis. In Saccharomyces cerevisiae, the AP-3 adaptor complex directs cargoes from the Golgi to the lysosomal vacuole. The AP-3 core consists of small and medium subunits complexed with two non-identical large subunits, β3 (Apl6) and δ (Apl5). The C-termini of β3 and δ were thought to be flexible hinges linking the core to ear domains that bind accessory proteins involved in vesicular transport. We found by computational modeling that the yeast β3 and δ hinges are intrinsically disordered and lack folded ear domains. When either hinge is truncated, AP-3 is recruited to the Golgi, but vesicle budding is impaired and cargoes normally sorted into the AP-3 pathway are mistargeted. This budding deficiency causes AP-3 to accumulate on ring-like Golgi structures adjacent to GGA adaptors that, in wild-type cells, bud vesicles downstream of AP-3 during Golgi maturation. Thus, each of the disordered hinges of yeast AP-3 has a crucial role in mediating transport vesicle formation at the Golgi.

publication date

  • November 1, 2024

has restriction

  • hybrid

Date in CU Experts

  • October 2, 2024 7:59 AM

Full Author List

  • Leih M; Plemel RL; West M; Angers CG; Merz AJ; Odorizzi G

author count

  • 6

Other Profiles

Electronic International Standard Serial Number (EISSN)

  • 1477-9137

Additional Document Info

volume

  • 137

issue

  • 21