From sarcomeric mutations to heart disease: Understanding familial hypertrophic cardiomyopathy
Journal Article
Overview
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Animal Population Groups - Mice, Transgenic
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Animals
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Aortic Valve Disease - Cardiomyopathy, Hypertrophic, Familial
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Biochemical Phenomena - Calcium Signaling
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Calcium-Binding Proteins - Calmodulin
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Cardiomyopathies - Cardiomyopathy, Hypertrophic, Familial
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Cell Physiological Phenomena - Calcium Signaling
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Female
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Genetic Diseases, Inborn - Cardiomyopathy, Hypertrophic, Familial
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Humans
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Ion Transport - Calcium Signaling
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Male
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Mice
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Mice - Mice, Transgenic
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Muscle Cells - Sarcomeres
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Muscle Cells - Sarcomeres
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Muscle, Striated - Sarcomeres
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Mutation
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Organelles - Sarcomeres
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Peptides - Calmodulin
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Peptides - Mitogen-Activated Protein Kinases
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Protein-Serine-Threonine Kinases - Mitogen-Activated Protein Kinases
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Proteins - Calmodulin
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Proteins - Mitogen-Activated Protein Kinases
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Sex Characteristics
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Transcription Factors
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Date in CU Experts
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September 6, 2013 4:09 AM
Full Author List
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Maass A; Konhilas JP; Stauffer BL; Leinwand LA
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