subject area of
- ACTIVATION OF ADULT RAT CNS ENDOTHELIAL CELLS BY OPIOID-INDUCED TOLL-LIKE RECEPTOR 4 (TLR4) SIGNALING INDUCES PROINFLAMMATORY, BIOCHEMICAL, MORPHOLOGICAL, AND BEHAVIORAL SEQUELAE Journal Article
- Activation of the medial prefrontal cortex by escapable stress is necessary for protection against subsequent inescapable stress-induced potentiation of morphine conditioned place preference Journal Article
- Brain-derived neurotrophic factor is essential for opiate-induced plasticity of noradrenergic neurons Journal Article
- Distinct patterns of ΔFosB induction in brain by drugs of abuse Journal Article
- Minocycline suppresses morphine-induced respiratory depression, suppresses morphine-induced reward, and enhances systemic morphine-induced analgesia Journal Article
- Stress potentiation of morphine-induced dopamine efflux in the nucleus accumbens shell is dependent upon stressor uncontrollability and is mediated by the dorsal raphe nucleus. Journal Article
- Stressor controllability modulates stress-induced dopamine and serotonin efflux and morphine-induced serotonin efflux in the medial prefrontal cortex Journal Article
- Surgical and pharmacological suppression of glucocorticoids prevents the enhancement of morphine conditioned place preference by uncontrollable stress in rats Journal Article
- The glial activation inhibitor AV411 reduces morphine-induced nucleus accumbens dopamine release Journal Article
- Uncontrollable stress potentiates morphine's rewarding properties Journal Article